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Caspofungin coadministration with cyclosporine showed a significant risk of hepatotoxicity. Other drug-drug interactions require dose adjustments p. 79 ; . The dose is 70 mg as a loading dose, followed by 50 mg once per day. NYSTATIN Mycostatin susp. and lozenges ; has fungistatic activity clinically limited to candidiasis moniliasis, thrush ; . It is poorly absorbed across any surface but is effective against cutaneous, oropharyngeal, and vaginal candidiasis that occasionally complicates broad-spectrum antibiotic therapy. No side effects or drug interactions occur. Dose: 1 tsp 5 ml ; qid pc. Swish in mouth, gargle, swallow. MICONAZOLE Monistat cream ; is useful as a skin or vaginal cream for candidal infections that may accompany broad-spectrum antibiotic therapy. It is available over the counter. GRISEOFULVIN Fulvicin, etc. ; provides systemic therapy against superficial dermatophyte infections of skin and hair; e.g., "ringworm." Do not use in pophyria patients. CLOTRIMAZOLE Lotrimin, et al. ; is for treatment of dermatological infections of tenia and candida types. Some cases of otomycosis may respond to use of the solution as ear drops. For treatment of oropharyngeal candidiasis, it is available without prescription ; as a troche Mycelex ; . Dose: dissolve in mouth 5 times daily. No adverse events or drug interactions occur. TERBINAFINE Lamisil ; is an oral antifungal for treatment of dermatophyte infections of the toenails or fingernails. Such infections have been thought to cause a secondary, allergic otitis externa in some patients. Improvement has been reported with prolonged oral administration of this agent: one 250 mg tablet daily for 6-12 weeks Oto. Clin. N.A. 1998; 31: 157 ; . Hepatotoxic potential makes pretreatment screening advisable. ACYCLOVIR Zovirax ; ointment is effective in the treatment of mucocutaneous Herpes simplex infections in immunocompromised patients. Intravenously or orally, it is effective against both localized and disseminated Herpes simplex and zoster infections i.e., Herpes zoster oticus ; .2 Orally it has suppressed or prevented symptomatic attacks of mucocutaneous Herpes simplex virus during the course of drug therapy e.g., 400 mg bid for 4 months ; . Unfortunately, herpes viruses persist in a latent form for prolonged periods e.g., neural cells of ganglia for Herpes simplex and zoster ; . Currently available herpes drugs require actively multiplying virus to be effective. Since none are active against latent virus, active infections can be expected to recur. Topical acyclovir is effective against Herpes simplex labialis, keratitis, and primary genital herpes. Renal dysfunction is encountered rarely with IV therapy it is reversible. Dosage for Herpes zoster: 800 mg q 4 hrs, 5 times daily for 7-10 days. For chicken pox adults and children over 40 kg ; : 800 mg qid for 5 days decreases severity of varicella if initiated within 24 hours of the rash. For Herpes simplex labialis: 400mg po 5 times daily q 4 hr while awake ; X 5 days. VALACYCLOVIR Valtrex, oral ; administered orally is rapidly converted into acyclovir at substantially higher serum levels. It has proven effective for shortening the course and discomfort of Herpes simplex labialis if it is initiated within 2 hours of symptom onset tingling, itching, burning ; . Dosage: 2 Gm po day optional additional 2 Gm po once on day 2 ; . Dosage for Herpes zoster: begin within 48 hours of rash, give 1 Gm tid for 7 days. For recurring Herpes simplex genital ; , begin within 48 hours of onset, 500 mg bid for 5 days. Section I.RAntivirals for ENT, H&N Infections.
Table of Contents Jennison does not file jointly with Prudential and, as such, shares reported on Jennison's Schedule 13G may be included in the shares reported in the Schedule 13G filed by Prudential which they were as described above ; . Jennison's address is 466 Lexington Avenue, New York, NY 10017. The information for Snyder Capital Management, L.P. and its general partner, Synder Capital Management Inc. collectively, the "Snyder Group" ; , is derived from a Schedule 13G filed with the SEC on February 14, 2007 by the Snyder Group which states, as of December 31, 2006, the Snyder Group beneficially owned 5, 701, 800 shares, with shared voting power with respect to 5, 148, 300 shares. Includes beneficial ownership of 131, 612 shares of restricted stock issued under our 2005 Plan and 606, 316 shares that are held by Michael J. Quillen, L.L.C. As of April 2, 2007, Ms. Bowers does not beneficially own any shares of Alpha's common stock. Includes beneficial ownership of 5, 035 shares of restricted stock issued under our 2005 Plan. For each of Messrs. Draper, Eisenberg and Fox, includes beneficial ownership of 3, 530 shares of restricted stock issued under our 2005 Plan. Also includes 4, 000 shares issuable upon exercise of options held by each of Messrs. Draper, Eisenberg and Fox, which are exercisable within 60 days of the date of this table. The stock amounts shown for Messrs. Draper and Fox do not include 3, 510 and 1, 325 share units, respectively, which were credited to their respective share unit accounts under the Deferred Compensation Agreement, which is described in more detail in "Additional Information Regarding Our Director Compensation Table." Includes beneficial ownership of 3, 530 shares of restricted stock issued under our 2005 Plan. Includes beneficial ownership of 47, 634 shares of restricted stock issued under our 2005 Plan and 41, 136 shares issuable upon exercise of options held by Mr. Brown that are exercisable within 60 days of the date of this table. Includes beneficial ownership of 73, 535 shares of restricted stock issued under our 2005 Plan and 29, 162 shares issuable upon exercise of options held by Mr. Crutchfield that are exercisable within 60 days of the date of this table. Includes beneficial ownership of 27, 721 shares of restricted stock issued under our 2005 Plan and 14, 368 shares issuable upon exercise of options held by Mr. Groves that are exercisable within 60 days of the date of this table. Includes beneficial ownership of 31, 609 shares of restricted stock issued under our 2005 Plan and 16, 000 shares issuable upon exercise of options held by Mr. Stuebe that are exercisable within 60 days of the date of this table. Includes beneficial ownership of 250, 000 shares subject to the security interest of Citigroup Global Markets Inc. under a pre-paid forward purchase agreement. Includes beneficial ownership of an additional 82, 761 shares of restricted stock issued under our 2005 Plan and 57, 849 shares issuable upon exercise of options that are exercisable within 60 days of the date of this table. Does not include the shares beneficially owned by Messrs. Brown and Kroh, who were not executive officers of Alpha on the date of this table.
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The occurrence of certain side effects may also be increased with the following medicines: - Medicines known as anti-arrhythmics such as amiodarone ; , used for the treatment of certain heart conditions - Medicines for the treatment or prevention of malaria - Antibiotic medicines, such as erythromycin - Diuretics water tablets, such as furosemide ; . The following medicines may reduce the effect of risperidone: - The herbal remedy St John's Wort Hypericum perforatum ; - Rifampicin an antibacterial medicine ; - Medicines for epilepsy such as phenytoin or carbamazepine If you start or stop taking such medicines you may need a different dose of risperidone. The following medicines may increase the effect of risperidone: - Quinidine used for treating certain types of heart disease ; - Medicines known as beta blockers, used for the treatment of certain heart conditions or high blood pressure - Antidepressants such as paroxetine or fluoxetine - Tegbinafine an antifungal medicine ; If you start or stop taking such medicines you may need a different dose of risperidone. Risperidone may alter the effect of medicines used for the treatment of high blood pressure such as phenoxybenzamine, labetalol, methyldopa and guanethidine ; . Please tell your doctor or pharmacist if you are taking, or have recently taken, any other medicines, including medicines obtained without a prescription. It is important that you tell your doctor everything about your condition and of any problems that you may have had in the past. For example, always tell your doctor if you are taking other medicines because taking some medicines together can be harmful. If you need an operation or an anaesthetic, tell the doctor or dentist that you are taking Risperidon Medis. Taking Risperidon Medis with food and drink Your tablets can be taken with or without food. You should avoid drinking alcohol whilst taking Risperidon Medis, as risperidone may strengthen the effect of alcohol and may make you feel drowsy. Pregnancy and breast-feeding Ask your doctor or pharmacist for advice before taking any medicine.
Than those infected with anthropophilic species. The first presenting symptoms will usually be small patches of scaly skin with a little hair loss. Once the hair and hair shafts are infected, they are weakened and can break off below the hair shaft, giving the appearance of black dots in the scalp. Patients in whom scalp ringworm is suspected should be referred to their GPs for tests eg, microscopy and culture of skin scrapings ; and antifungal therapy. Treatment Tinea capitis must be treated with systemic antifungal agents because topical agents cannot penetrate all the infected areas. The length of treatment is determined by the time it takes for infected hair and skin to grow out and this varies according to season. Treatment is deemed successful when culture of skin scrapings from the infected area shows no growth of the organism involved. Systemic antifungal medicines include griseofulvin, itraconazole, ketoconzole, fluconazole and terbinafine. Griseofulvin is effective against T tonsurans and Microsporum species. Oral ketoconazole and itraconazole are usually avoided because of the risks of liver toxicity and fluconazole is not licensed for this indication. Oral terbinafine is not licensed for use in children but there is wide experience of use for this indication and dosage instructions are given in the British National Formulary for children over the age of one year. Treatment is usually for two weeks. Griseofulvin is the only licensed treatment for tinea capitis in children of one month and older. An oral daily dose of 1g for up to three months is indicated for adults and 1520mg per kg for children under 50kg. Note, this is higher than the dose recommended by Prodigy. ; A suspension is available from specialist importers. Griseofulvin has many side-effects that affect about 20 per cent of patients. These include headache and gastrointestinal upset and, more rarely, photosensitivity and lupus-like symptoms. It is contraindicated in several circumstances including severe liver disease and pregnancy -- pregnancy should be avoided during and for one month after treatment, and men should be advised not to father children within six months of treatment. Antifungal shampoos are believed to be useful when used in conjunction with systemic treatment to reduce transmission of the disease through shedding of spores. For the same reasons, some dermatologists recommend treating close contacts who may be asymptomatic carriers. Suitable shampoos are those containing selenium sulphide, ketoconazole or povidone iodine.
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1 Druker BJ, Lydon NB. Lessons learned from the development of an abl tyrosine kinase inhibitor for chronic myelogenous leukemia. J Clin Invest 2000; 105: 37. Druker BJ, Talpaz M, Resta DJ et al. Efficacy and safety of a specific inhibitor of the BCR-ABL tyrosine kinase in chronic myeloid leukemia. N Engl J Med 2001; 344: 10311037. Druker BJ, Tamura S, Buchdunger E et al. Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells. Nat Med 1996; 2: 561566. Deininger MW, Goldman JM, Lydon N et al. The tyrosine kinase inhibitor CGP57148B selectively inhibits the growth of BCR-ABL-positive cells. Blood 1997; 90: 36913698. Gambacorti-Passerini C, le Coutre P, Mologni L et al. Inhibition of the ABL kinase activity blocks the proliferation of BCR ABL + leukemic cells and induces apoptosis. Blood Cells Mol Dis 1997; 23: 380394. Buchdunger E, Cioffi CL, Law N et al. Abl protein-tyrosine kinase inhibitor STI571 inhibits in vitro signal transduction mediated by c-kit and platelet-derived growth factor receptors. J Pharmacol Exp Ther 2000; 295: 139145. Heinrich MC, Griffith DJ, Druker BJ et al. Inhibition of c-kit receptor tyrosine kinase activity by STI 571, a selective tyrosine kinase inhibitor. Blood 2000; 96: 925932. Okuda K, Weisberg E, Gilliland DG et al. ARG tyrosine kinase activity is inhibited by STI571. Blood 2001; 97: 24402448. Carroll M, Ohno-Jones S, Tamura S et al. CGP 57148, a tyrosine kinase inhibitor, inhibits the growth of cells expressing BCR-ABL, TEL-ABL, and TEL-PDGFR fusion proteins. Blood 1997; 90: 49474952 and
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Fda public health advisory about sporanox and lamisil ii the safety of sporanox capsules and lamisil tablets for the treatment of onychomycosis the food and drug administration fda ; is issuing a public health advisory concerning sporanox itraconazole ; capsules and lamisil terbinafine hydrochloride ; tablets for the treatment of onychomycosis.
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Tinea corporis Fig. 4 ; has the characteristic annular features normally associated with ringworm. The plaques are erythematous and scaly, with a well-defined active edge. This edge becomes more prominent as the infection resolves and the centre clears. Vesicles and pustules may be present in severe infections. The eruptions can be confused with eczema. However, unlike eczema, ringworm is contagious and will spread rapidly among children when toys are shared. A topical antifungal such as miconazole or terbinafine will usually eradicate the infection. Carriers are often asymptomatic. When ringworm is diagnosed, a person may feel very selfconscious, embarrassed, ashamed or dirty. The nurse will need to respond appropriately to any anxieties and explain to the patient how he or she can limit cross-infection through good hygiene practices. Tinea vesicolor can also affect the trunk and limbs, but is mainly found on the chest, back and shoulders. The causative organism is not a dermatophyte, but the resident yeast pityrosporum. At first glance, the condition could be confused with vitiligo. On dark skin, round hypopigmented patches occur, whereas on pale skin there are hyperpigmented lesions Lesher et al 1994 ; . Tinea vesicolor responds well to selenium sulphide shampoo; oral antifungals are seldom required. Unlike Tinea corporis, this condition is not considered to be contagious. This fact, however, should not detract from the need to be sensitive to the psychological impact of the presenting features. A tinea infection should not be confused with a candida infection. Candida is a resident yeast of the oral mucosa, gastrointestinal tract and vagina. The yeast thrive in warm, moist, occlusive.
A search in the Medline database revealed several publications concerning the association of terbinafihe with both exacerbations of pre-existing psoriasis, as well as de novo development of psoriasis. The skin lesions generally resolved within one month of terbinfine discontinuation, sometimes with psoriasis treatment.[3-5] and valaciclovir.
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Table 1 Variable CWT Cystic fibrosis growth parameters in Saudi population. n % ; 65 13 CHT 38 15 48 Percentile 75% 75-79% 80-84% Severe stunted Moderately stunted Mildly stunted Normal Figure 1 - WHZDX - weight for height Z-score at diagnosis, WHZ6 weight for height Z-score at 6 months follow up, WHZ12 weight for height Z-score at 12 months follow up, WHZ18 weight for height Z-score at 18 months follow up, WHZ24 weight for height Z-score at 24 months follow up. Zero line determine the mean weight Z-score of the standard population, -1 line determine the mean weight Z-score of the study population. Table 3 - Culture results of cystic fibrosis patients. N 190 ; Table 2 - Weight for height Z-score during 2 years follow up. Organism Period WHZ at diagnosis WHZ at 6 months WHZ 12 months Z score 12 and 6 months WHZ 18 months Z score 18 and 12 months WHZ 24 months Z score 24 and 18 months Mean score -1.7 0.16 -0.77 2 -0.86 1.01 -0.89 1.01 -0.93 1.01 n 74 a first culture after diagnosis b follow up culture, which is taken 1-6 month after diagnosis p-value 0.0001 Staphylococcus aureus Hemophilus influenzae Pseudomonas aeruginosa Streptococcus pneumonia Methicillin resistant Branhamella catarrhales Gram negative rods Gram positive cocci Virus Other 29 32 83 First culture 190 ; b ; Follow up 160 ; n % ; n % ; Status Severe malnutrition Moderate malnutrition Mid malnutrition Under weight Normal weight, for example, lamisil terbinafine.
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1188-1193 6 ; publisher: blackwell publishing previous article next article view table of contents key: - free content - new content - subscribed content - free trial content abstract: background oral terbinafine and oral itraconazole are two of the most common agents used for the treatment of toenail dermatophyte onychomycosis and
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Rifabutin Restricted to patients with mycobacterial infections resistant to conventional anti-tuberculosis drugs. Rifater Rifinah Rimactazid Streptomycin Dapsone 5.1.12 Ciprofloxacin Excluding eye drops and 100mg tablets for uncomplicated UTI. Oral ciprofloxacin has good bioavailability and should be prescribed in preference to IV whenever possible. Ofloxacin Restricted to patients with pelvic inflammatory disease. Levofloxacin Restricted to second line use by hospital specialists for penicillin allergic patients with community acquired pneumonia or for cystic fibrosis patients intolerant of ciprofloxacin where a quinolone is required. Moxifloxacin Restricted to second line use by hospital specialists for penicillin allergic patients with community acquired pneumonia or for cystic fibrosis patients intolerant of ciprofloxacin where a quinolone is required. Trimethoprim First line choice for uncomplicated UTIs. Nitrofurantoin 5.2 Fluconazole Griseofulvin Itraconazole Not approved for fungal nail infections. Terbinafine!
Side effects Whether or not you experience side effects will depend on how much medication you take and how long you take it for. With short-term use, people can sometimes experience general effects of brain slowing such as tiredness, blurring of vision, mild dizziness, slurred speech and mild short-term memory loss. Some people find that benzodiazepines affect their emotional state and they can feel a bit confused, irritable at times and possibly depressed. These general slowing effects make it dangerous for people to drive or operate machinery. With longer-term use people can describe feeling generally unmotivated or apathetic, increasingly irritable and, again, depressed. They may have headaches. Dependency or addiction As described above, either physical or psychological dependence can occur. This means that people end up taking the drug for longer than they meant to and in higher doses than was originally intended. When they try to control their drug use, people find that they are unable to do so. Use of the drug becomes a central part of the person's life and a good deal of time is spent thinking about the drug, making sure that an adequate supply is available, and planning activities around the taking of the drug. If a withdrawal state occurs, a number of symptoms of brain over-activity can be experienced remember that benzodiazepines work by slowing brain function - if the slowing is taken away, the brain rebounds and switches to an overactive state ; . Symptoms of withdrawal include and
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Just over six years ago, a small group of dedicated individuals decided that southeastern Wisconsin should have an Affiliate of the Susan G. Komen Breast Cancer Foundation. These people had seen the Race for the Cure in other cities and were convinced that southeastern Wisconsin was fertile ground for a similar event and all that went with it. Since 1999, many milestones have occurred for our Milwaukee Affiliate. We were incorporated in September of 1999. We have seen our Race for the Cure go from infancy to adulthood. Other events, such as the Bowl for the Cure and Lee Denim Day, have taken hold in the communities we serve. We have, since our inception, granted over $1 million to organizations that provide breast health education, screening and treatment. This newsletter is another milestone in our Affiliate's development. We are excited to establish the newsletter as a vehicle for communication and inspiration. Welcome to the Inaugural Newsletter! - Lorna Granger, Affiliate President.
1. Identification--Bancroftian filariasis is an infection with the nematode Wuchereria bancrofti, which normally resides in the lymphatics in infected people. Female worms produce microfilariae that reach the bloodstream 6 12 months after infection. Two biologically different forms occur: in one, the microfilariae circulate in the peripheral blood at night nocturnal periodicity ; with greatest concentrations between 10 and 2 am; in the other, microfilariae circulate continuously in the peripheral blood, but occur in greater concentration in the daytime diurnal ; . The latter form is endemic in the South Pacific and in small rural foci in southeastern Asia where the principal vectors are day-biting Aedes mosquitoes. Clinical manifestations in regions of endemic filariasis include: a ; asymptomatic and parasitologically negative form; b ; asymptomatic microfilaraemia; c ; filarial fevers manifested by high fever, acute recurrent lymphadenitis and retrograde lymphangitis with or without microfilaraemia; d ; lymphostasis associated with chronic signs, including hydrocoele, chyluria, lymphoedema and elephantiasis of the limbs, breasts and genitalia, with low-level or undetectable microfilaraemia; and e ; tropical pulmonary eosinophilic syndrome, manifested by paroxysmal nocturnal asthma, chronic interstitial lung disease, recurrent low-grade fever, profound eosinophilia and degenerating microfilariae in lung tissues but not in the bloodstream occult filariasis ; . Brugian and Timorean filariasis are caused by the nematodes Brugia malayi and B. timori respectively. The nocturnally periodic form of B. malayi occurs in rural populations living in open rice-growing areas throughout much of southeastern Asia. The subperiodic form infects humans, monkeys and wild and domestic carnivores in the forests of Malaysia and Indonesia. Clinical manifestations are similar to those of.
Signs of toenail fungus include thickening, yellow or whitish discoloration and flaking or crumbling of the nail. The least expensive way of confirming the diagnosis is by having a potassium hydroxide test performed. This test is done by taking a scraping of the involved nail and then treated it with potassium hydroxide. This chemical dissolves everything in the sample except for the fugal elements which can then be identified through a microscope. Once the infection is confirmed, there are a number of treatment options available. Unfortunately, the over-the-counter antifungal nail creams and ointments are minimally effective. The most effective treatment generally involves the use of a prescription antifungal medication that is taken by mouth. Even the prescription topical nail lacquer only works for about 1 in 10 cases. Many experts consider the medication terbinafine Lamisil ; to be the treatment of choice for these infections. Treatment usually takes several months and during this time, it is important to have the doctor monitor for side effects of the medication such as liver dysfunction. Other effective medications include itraconazole Sporanox ; and fluconazole Diflucan ; . The American Podiatric Medicine Society : apma ; offers the following tips for preventing toenail fungal infections.
Comment references in may 2000 in the netherlands, the manufacturer of terbinafine, novartis, started a nationwide information campaign which included television advertisements advising people with onychomycosis to visit their general practitioner.
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2. acetic acid$ or acetate$ or acetamide$ or acetoxyacetylaminofluorene$ or hydroxyacetylaminofluorene$ or allylisopropylacetamide$ ; .ti, ab. 3. idoacetamide$ or idoacetate$ or piracetam$ or thioacetamide$ or gadolinium$ or technetium$ or dichoroacetate$ or fluoroacetate$ or iodoacetate$ ; .ti, ab. 4. foscarnet$ or thioglycolate$ or acetic anhydride$ ; .ti, ab. 5. aminooxyacetic or edetic or egtazic or iodoacetic or nitrilotriacetic or pentetic or peracetic or phosphonoacetic or trichloroacetic or trifluoroacetic ; adj acid$ ; .ti, ab. 6. exp ANTIFUNGAL AGENTS 7. therapeutic fungicide$ or antifungal agent$ or antifungals ; .ti, ab. 8. benzoate$ or butenafine$ or chlorquinaldol$ or cyclosporine$ or dichlorophen$ or fluconazole$ or flucytosine$ or glycyrrhizic acid$ or hexetidine$ or itraconazole$ or monensin$ or nifuratel$ or pentamidine$ ; .ti, ab. 9. co-amoxiclav$ or sodium benzoate$ or thimerosal$ or thiram$ or thymol$ or tolnaftate$ or tomatine$ or triacetin$ or trimetrexate$ ; .ti, ab. 10. amoroldine$ or benzoic acid$ or clotrimazole$ or econazole$ or ketoconazole$ or miconazole$ or nystatin$ or Salicylic acid$ or sulconazole$ or terbinafine$ or tioconazole$ or undecenoate$ ; .ti, ab. 11. antiviral$ or anti viral$ or idoxuridine$ ; .ti, ab. 12. acetylcysteine$ or acyclovir$ or amantadine$ or aphidicolin$ or aprotinin$ or brefeldin or bromodeoxyuridine$ or cytarabine$ or deoxyglucose$ or dextran sulfate$ ; .ti, ab. 13. dideoxyadenosine$ or dideoxynucleoside$ or dihematoporphyrin ether$ or ditiocarb$ or filipin$ or floxuridine$ or ganciclovir$ or inosine pranobex or interferon alfa$ or interferon type$ or interferon beta or interferon gamma or interferons ; .ti, ab. 14. methisazone$ or phosphonoacetic acid$ or poly a-u or poly i-c or pyran copolymer$ or ribavirin$ or rimantadine$ or streptovaricin$ or tenuazonic acid$ or tilorone$ or trifluridine$ or tunicamycin$ or vidarabine$ ; .ti, ab. 15. exp BACITRACIN 16. exp Povidone-Iodine 17. bacitracin$ or povidone iodine$ or betaisodona$ or polyvinylpyrrolidone iodine$ or betadine$ or disadine$ or isodine$ or pvpi or pharmadine$ ; .ti, ab.
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Sinus rhythm was established using computerized analysis system. The heart rate variability was investigated after atrial fibrillation or atrial flutter cadiovertion to the sinus rhythm. All patients were divided into two groups: 1 group, 16 patients to whom the sinus rhythm was restored using antiarrhythmic drugs and 2 group, 27 patients to whom sinus rhythm was restored by electrical defibrillation. For the first group patients long-term ECG was recorded three times: before and during sinus rhythm restoration period as well as immediately after sinus rhythm restoration at least 12 hour or mostly 812 hours duration. And for second one - long-term ECG was recorded twice: 0.51 hour period before and 0.51 hour period after sinus rhythm restoration. Results and Discussion Individual type of heart rhythm disturbances can be easily identified by Poincare plot of RR intervals, which have got specific patterns for any of them. An atrial fibrillation seems like "a fan" Fig. 1.
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